Introducing solid foods is a very important step in your baby’s development and well-being. In fact, studies show that babies who are fed nutritious, healthy diets grow into stronger kids and better-adjusted eaters than those who are fed poor diets.
Many parents don’t realize that making baby food at home is a simple and economical plan to provide your baby with the best in quality, nutrition and taste. It makes it easy for you to ensure your child gets the best start possible. Making baby food using fresh, all-natural ingredients at home has many benefits, including:·
Increased nutritional value·
Elimination of additives·
Improved freshness ·
Added variety·
Enhanced control·
Lower costsRead more:
Vitamins and other nutrients are critically important to your baby. For the next three years, your baby will experience rapid growth and development. It is essential that he be fed a healthy and nutritious diet to maximize his growth and development process. Processed baby foods have added water, sugars and starchy fillers. While these products are not nutritionally bad for your baby, their use in baby food dilutes the nutrient content of the actual foods. To make matters worse, processed baby foods are cooked at high temperatures to kill bacteria, so they can be stored in jars at room temperature. Bacteria are not the only things that are eliminated in this process. Vitamins and nutrients are also destroyed. Many baby food manufacturers compensate for the loss of vitamins by artificially adding some of them back in after the food is processed.When you make baby food at home, you can cook it quickly. This process not only preserves the wonderful color and taste of the food, but most importantly it maximizes the foods’ nutrient content for your precious baby.
Processed baby foods contain trace amounts of chemicals, including pesticides, herbicides and fungicides. Although the U.S. Food and Drug Administration has approved these chemicals, you may choose not to feed your baby products containing them. Buying certified organic produce (fresh or frozen) and preparing food at home eliminates agricultural chemicals from your baby’s diet. In addition, many varieties of processed baby foods add ingredients that are not essential or beneficial to your baby’s diet. These can include ingredients such sugar, butter and salt. Most healthcare professionals will recommend you avoid the introduction of these foods until your child is much older. Homemade baby food is pure, wholesome food with nothing added that you did not add yourself.Improved freshness (homemade baby food vs bought food)Have you compared fresh green peas to a jar of pea baby food? Even though they are the same food, they don’t look, smell or taste similar. While your baby does not have the refined palate of an adult, he does respond to taste, color and smell. With the enormous availability of fresh produce in your grocery store and the simplicity of making baby food, there is really is no reason he needs to be deprived of colorful, tasty, great-smelling baby food. And serving fresh food from the very beginning will help your baby be more open to tasting new flavors and types of food. Read more:
Processed baby food is developed for the mass market and, as a result, is limited in variety. Variety is key to a balanced diet and healthy living. Today’s grocery stores offer a tremendous variety of fresh and frozen fruits and vegetables. There is no reason why your baby should be limited by what food manufacturers consider the most popular foods. What’s more, preparing baby food at home enables you to add herbs, combine flavors, and easily introduce new textures, making your baby’s mealtime a pleasurable, gourmet experience.
As a parent, you want to understand and trust the ingredients in your baby’s diet. Similarly, you want assurance concerning the purity, safety, quality and consistency of such ingredients. Preparing baby food at home provides you with control of your baby’s diet and knowledge of exactly what goes into your baby’s food. The more involvement you have with what you are feeding your baby, the more likely you are to nurture healthy eating habits.
http://www.familyresource.com/pregnancy/nutrition/making-baby-food-homemade-babyfood#ixzz0mSW2sjBN
Thursday, April 29, 2010
Monday, April 26, 2010
Hemophilia in Malaysia
The Hemophilia Society of Malaysia organizes training workshops for patients with support from the World Federation of Hemophilia (WFH) and Baxter Healthcare.
IMAGINE having a condition where you could end up bleeding to the point where you suffer from joint deformity, all because of a simple knock or bump. This is the reality of a person living with hemophilia.
Hemophilia is not as rare as you might think, but very few people know much about it. There are so many myths about this condition, and people who live with it not only have to deal with daily adjustments, they also need to educate the people around them.
Hemophilia is a life-long bleeding disorder that prevents blood from clotting properly due to a lack of clotting factor. This results in prolonged bleeding; hence simple cuts and wounds can have severe and debilitating consequences for individuals with hemophilia, especially when bleeding occurs internally.
Hemophilia is a genetic condition carried on the X chromosome and therefore passed on by women. With each pregnancy, a woman who is a carrier of hemophilia has a 25% chance of giving birth to a son with hemophilia.
Since the genetic make-up of the father’s sperm determines the sex of the baby, all the daughters of a man with hemophilia will be carriers of the condition, carried on the X chromosome, but none of his sons.
In about 30% of cases, there is no family history of the disorder and the condition is the result of a spontaneous genetic mutation.
The most severe forms of hemophilia affect almost only males. Although extremely rare, a female can be affected if her father has hemophilia and her mother is a carrier. Many women who are carriers can also show symptoms of mild hemophilia.Two of the most common forms of hemophilia are A and B, with hemophilia A being the more typical form. More than 400,000 people in the world have hemophilia. In Malaysia, there are approximately 900 people affected by hemophilia A and another 200 affected by hemophilia B.
People living with hemophilia do not have enough of, or are missing, one of the blood clotting proteins naturally found in blood.
Clotting factors, designated by Roman numerals I through XIII, are proteins found in blood that work together to make blood clot. When the body detects bleeding, clotting factors are switched on in a specific order, each activating the next factor in the "blood clotting cascade" to ultimately form a fibrin clot. The fibrin clot acts like a net, holding the platelets together to create a firm blood clot.
In people with hemophilia A, clotting factor VIII is not present in sufficient amounts or is absent. Factor VIII is one of the clotting factor proteins that help produce the fibrin clot. In such people, firm blood clots do not form in the wound and bleeding is not easily stopped. Without enough factor VIII, patients can experience spontaneous, uncontrolled internal bleeding that is painful, debilitating and damaging to joints.
If untreated, patients with severe hemophilia A have a greatly reduced life expectancy.
"The training workshops that are being held are a crucial form of on-going education in managing and dealing with hemophilia, because it is possible for individuals with the condition to live relatively normal and healthy lives when proper treatment and care is administered," explained Mohamed Aris Hashim, President of the Hemophilia Society of Malaysia.
Hemophilia is treated by replacing the missing clotting factor in the blood via an injection containing the needed factor. Bleeding stops when enough clotting factor reaches the bleeding site.
Treatment for hemophilia has evolved tremendously over the past few decades. The development of plasma concentrates was a breakthrough that allowed patients to stop their bleeding via a simple injection that could be self-administered. However, concerns with contracting blood-borne infections such as hepatitis B, hepatitis C and HIV continued to be a problem.
It was not until the 1980s that genetically engineered factor concentrates were developed, leading to the very recent and revolutionary innovation of a recombinant factor VIII treatment that is free from human plasma and animal-derived albumin which eliminates the risk of pathogen transmission.
"The greatest challenge for people with bleeding disorders in many countries is access to safe and affordable treatment. WFH is working to achieve our vision of treatment for all, which includes improved access to clotting factor concentrates. Besides that, it also means proper diagnosis, management, and care provided by an interdisciplinary team of trained specialists," said Mark Skinner, President of the World Federation of Hemophilia and a hemophilia patient.
The full day workshop, which was divided into separate sessions for men, women and children to address the different challenges each are faced with, was held at Hospital Ampang and attended by 150 participants. Topics included Raising a Child with Hemophilia, fitness for women and men as well as group discussions on the emotional adjustments that are necessary in a family with hemophilia.
"Living with hemophilia in the family is a constant learning process, hence family and social support is one of the main reasons why the society exists," explained Aris. "We’ve found that having like-minded individuals sharing their stories and experiences is like having a very large extended family, and all our activities, such as this workshop, are aimed at helping each other live our best lives possible.”
The Hemophilia Society of Malaysia was established in 1980 specifically to promote the welfare and to help patients and their families in overcoming the myriad of problems associated with this potentially crippling condition.
It has been regularly bringing together families from various parts of Malaysia to participate at educational workshops and camps, producing educational materials and promoting blood donation campaigns.
The Ministry of Health Malaysia has accredited the society as the official representative of the hemophilia and other bleeding disorders community in the country. The society is also affiliated with the World Federation of Hemophilia that has 115 member countries worldwide.
IMAGINE having a condition where you could end up bleeding to the point where you suffer from joint deformity, all because of a simple knock or bump. This is the reality of a person living with hemophilia.
Hemophilia is not as rare as you might think, but very few people know much about it. There are so many myths about this condition, and people who live with it not only have to deal with daily adjustments, they also need to educate the people around them.
Hemophilia is a life-long bleeding disorder that prevents blood from clotting properly due to a lack of clotting factor. This results in prolonged bleeding; hence simple cuts and wounds can have severe and debilitating consequences for individuals with hemophilia, especially when bleeding occurs internally.
Hemophilia is a genetic condition carried on the X chromosome and therefore passed on by women. With each pregnancy, a woman who is a carrier of hemophilia has a 25% chance of giving birth to a son with hemophilia.
Since the genetic make-up of the father’s sperm determines the sex of the baby, all the daughters of a man with hemophilia will be carriers of the condition, carried on the X chromosome, but none of his sons.
In about 30% of cases, there is no family history of the disorder and the condition is the result of a spontaneous genetic mutation.
The most severe forms of hemophilia affect almost only males. Although extremely rare, a female can be affected if her father has hemophilia and her mother is a carrier. Many women who are carriers can also show symptoms of mild hemophilia.Two of the most common forms of hemophilia are A and B, with hemophilia A being the more typical form. More than 400,000 people in the world have hemophilia. In Malaysia, there are approximately 900 people affected by hemophilia A and another 200 affected by hemophilia B.
People living with hemophilia do not have enough of, or are missing, one of the blood clotting proteins naturally found in blood.
Clotting factors, designated by Roman numerals I through XIII, are proteins found in blood that work together to make blood clot. When the body detects bleeding, clotting factors are switched on in a specific order, each activating the next factor in the "blood clotting cascade" to ultimately form a fibrin clot. The fibrin clot acts like a net, holding the platelets together to create a firm blood clot.
In people with hemophilia A, clotting factor VIII is not present in sufficient amounts or is absent. Factor VIII is one of the clotting factor proteins that help produce the fibrin clot. In such people, firm blood clots do not form in the wound and bleeding is not easily stopped. Without enough factor VIII, patients can experience spontaneous, uncontrolled internal bleeding that is painful, debilitating and damaging to joints.
If untreated, patients with severe hemophilia A have a greatly reduced life expectancy.
"The training workshops that are being held are a crucial form of on-going education in managing and dealing with hemophilia, because it is possible for individuals with the condition to live relatively normal and healthy lives when proper treatment and care is administered," explained Mohamed Aris Hashim, President of the Hemophilia Society of Malaysia.
Hemophilia is treated by replacing the missing clotting factor in the blood via an injection containing the needed factor. Bleeding stops when enough clotting factor reaches the bleeding site.
Treatment for hemophilia has evolved tremendously over the past few decades. The development of plasma concentrates was a breakthrough that allowed patients to stop their bleeding via a simple injection that could be self-administered. However, concerns with contracting blood-borne infections such as hepatitis B, hepatitis C and HIV continued to be a problem.
It was not until the 1980s that genetically engineered factor concentrates were developed, leading to the very recent and revolutionary innovation of a recombinant factor VIII treatment that is free from human plasma and animal-derived albumin which eliminates the risk of pathogen transmission.
"The greatest challenge for people with bleeding disorders in many countries is access to safe and affordable treatment. WFH is working to achieve our vision of treatment for all, which includes improved access to clotting factor concentrates. Besides that, it also means proper diagnosis, management, and care provided by an interdisciplinary team of trained specialists," said Mark Skinner, President of the World Federation of Hemophilia and a hemophilia patient.
The full day workshop, which was divided into separate sessions for men, women and children to address the different challenges each are faced with, was held at Hospital Ampang and attended by 150 participants. Topics included Raising a Child with Hemophilia, fitness for women and men as well as group discussions on the emotional adjustments that are necessary in a family with hemophilia.
"Living with hemophilia in the family is a constant learning process, hence family and social support is one of the main reasons why the society exists," explained Aris. "We’ve found that having like-minded individuals sharing their stories and experiences is like having a very large extended family, and all our activities, such as this workshop, are aimed at helping each other live our best lives possible.”
The Hemophilia Society of Malaysia was established in 1980 specifically to promote the welfare and to help patients and their families in overcoming the myriad of problems associated with this potentially crippling condition.
It has been regularly bringing together families from various parts of Malaysia to participate at educational workshops and camps, producing educational materials and promoting blood donation campaigns.
The Ministry of Health Malaysia has accredited the society as the official representative of the hemophilia and other bleeding disorders community in the country. The society is also affiliated with the World Federation of Hemophilia that has 115 member countries worldwide.
IHSAN:2nd Hospitalization
Part 1,Date:9.4.2010
Pagi ni aku,husben n ihsan bersiap awal sbb kami nak bawa ihsan ke Klinik Pakar Kanak2 An-Nur atas masalah lebam2 kt badan ihsan.Dalam pukul 7 lebih kami dah kuar.Tp klinik bukak pukul 9am,jadi kami pi breakfast dulu kt Kopitiam berhampiran.Selesai makan terus zasss ke klinik berkenaan.
Kt dlm klinik tu ada byk mainan,jadi sajalah bagi ihsan duduk kt mainan kuda,suka sgt ihsan.Sementara nk tunggu turn kami,ihsan tgk plak ada dua org kanak2 yg agak besar dari dia main ape ek nama mainan tu xingat plak.Bukan main suka ihsan tgk abang dan kakak tu smpai gelak2 terkekek padahal diorang tak kacau pun ihsan.Tiba2 turn ihsan jumpe Dr,xingat plak nama Dr ni tapi Dr pompuan la,baik sgt lembut je.
Lepas dah terangkan mslh ihsan tu Dr check ihsan and the terus nk amik darah nk tgk ape mlsh ihsan.So time nk amik darah ihsan biasalah sure nangis.Tp aku respect ah Dr ni sbb dia amik darah ihsan kejap je n tak bengkak pun.Tp ihsan tetap nangislah kan for sure.Dr suh tunggu kat luar sementara tunggu result.Kami pun tunggu la kt luar,aku kata kt husben td gelak2 ihsan skang kan dh nangis.Pastu aku pun susukan ihsan,naseb baik kt situ ada bilik utk menyusu.
Tak lama lepas tu nurse panggil kami.Dr kata hemoglobin ihsan low 7.4.Dr kata kalu ibu menyusu ramai je yg datang anaknye hemoglobin low dlm 5 cmtue tp sbb ihsan ada bruises so Dr suh bawak pi HUKM terus.Dr terus buat surat.
Aku pun terus call HUKM (tingkat 7) nk tnye ada tak Dr hari ni utk jmpe ihsan.Nurse tu tnye mcm2 ape sakit ihsan sumer,last dia kata nnt jmpe Prof Asma dlm pukul 1245pm sbb dulu ihsan pernah masuk wad under Prof Asma atas mslh cervical lymphoside (betul ke eja?) nye tuh.Sblm pi sana smpat jugak aku singgah ke Butik Ariani yg berdekatan past uterus singgah umah mak jap sbb dekat je dari umah mak nk pi HUKM tu.
Dalam 1215pm kami pun gerak dari umah mak.Terus naik ke tingkat 7 n tunggu utk jmpe Prof Asma.Dlm pukul 1pm baru dpt jmpe nasib Prof Asma sudi meluangkan masa utk tgk ihsan.TQ Prof Asma,.Lepas tgk2 ihsan Prof pun kata “kalu sy kata ihsan kena masuk wad….aku pun sengih je la kt husben sbb masa dlm keta ada tercakap mcm tu,kalu ihsan kena masuk wad cmne….alih2 betul la pulak.Prof Asma nk call Prof B tp mcm takut2 je,katanye PAED ni time lunch hour xangkat call so Prof sms je dulu.Aku mcm pelik Prof Asma cm takut je ngan Prof B rupe2nye semua nurse pun takut gak kt Prof B sbb dia HOD kt PAED bawah tu.huhu.
Then after Prof B balas sms Prof Asma terus Prof Asma call n bgtau condition ihsan.Tp Prof B nk ihsan masuk wad kt bawah bukan kt atas sbb hari sabtu/ahad tak de Dr nk tgk kt tingkat 7 tuh.emmm…so aku ngan husben pun ok je la demi kesihatan ihsan.Jadi hari itu sekali lagi ihsan kena masuk,Bila dh keluar dari bilik Prof jam pun dh masuk waktu zohor terusla husben ke surau bawah utk solat jumaat,tinggallah aku ngan ihsan kt situ sementara tunggu husben ku.
Dlm pukul 2pm husben pun dtg balik,aku kata nurse kata pi daftar dulu kt bawah,tp xde katil kosong lagi.So aku decide nk balik umah dulu kemas2 barang sumer sbb xtau baper lame plak ihsan nk kena tahan kt wad ni.
Dalam pukul 515pm kami pun smpai kt WAD 4A,katil 9.Bermulalah episode baru ihsan di sini.Ptg tu mak n abah pun ada dtg.Husbenku pulak dh gi kelas kt UPM itupun dlm pukul 630pm dia baru gerak.Dlm pukul 7pm mak n abah pun balik.Tinggal la ku berdua ngan ihsan kt sini.Dalam pukul 7 lebih ada sorang Dr pompuan datang. Dr Azlynn ke xsilap namanye mcm2 dia tnye pasal ihsan ni.Pening kepalaku nk menjawab mcm pesalah la pulak nasib ihsan mood ok masa tu.Dr check ihsan pun pening sbb ihsan xlalu diam kaki tangan tiba2 Dr dpt call emergency so dia terpaksa gi dulu.
Tak lama ihsan nenen n tido.Datang pulak Dr lelaki katanye nk tnye mcm2 la.Dr tu tnye mcm2 gak mcm Dr tadi tu tapi kali ni dia tulis balik dlm record ihsan.Pastu tak lama dating pulak 2-3 orang Dr sama gak tnye mcm2 pasal ihsan n check lebam2 ihsan tu.Dr tu suh amik darah ihsan malam ni gak utk tgk ape mlsh nye.Pastu dlm pukul 10pm aku sms husben tnye tak nak dtg ke,aku lapar nk makan.So husben pun dtg bawak makanan.
Aku asyik dengar bunyi baby nangis tp aku xtau dari mana,pastu husben ku kata kt sblh ni baby tux de orang tgk pun.Yeke aku kata???terus aku pi sblh mmg baby tu yg nangis sian sgt aku tgk,dh la asma batuk plak tu.Kakak yg sebelah katil ngan baby tu bgtau ni anak kebajikan xde mak ayah.Ya ALLAH siannye aku ngan baby yg diberi nama ARISYA ini.Terseksa aku tgk dia batuk,aku rasa mungkin dia lapar.Tapi xada nurse yg dtg tgk sbb malam tu nurse ada 3 orang je pastu ada case emergency plak.Aku dukung la arisya kejap,dia diam.Pastu aku bagi dia mnm air masak guna botol ihsan tgk kalu2 dia lapar.Mmg dia lapar,sian sgt.Aku suh husben jaga arisya kejap sbb aku nk pump susu bagi kat arisya.Alhamdulillah ada rezeki arisya utk mnm susu aku.Syukur padamu ALLAH yg maha kaya.Lps tu dia diam maybe sbb da kenyang.Aku rasa dh lewat malam baru nurse amik arisya tukar pampers n bagi susu tp arisya mcm dh tak nak sbb tadi dia dh dpt susu ibu.
Pastu dlm pukul 11 lebih ke husben balik dia takut nak tdo sini esok malam baru tdo ye lah takut kena marah ngan nurse.Then dlm pukul 12 lebih Dr panggil nk amik darah ihsan.Aku tau sure ihsan menjerit2 la lepas ni.Sian sgt kt ihsan.Okla nnt aku smbg lagi sbb panjang lagi kisah di WAD 4A sepanjang 7 hari kami berkampung kt sini.
Pagi ni aku,husben n ihsan bersiap awal sbb kami nak bawa ihsan ke Klinik Pakar Kanak2 An-Nur atas masalah lebam2 kt badan ihsan.Dalam pukul 7 lebih kami dah kuar.Tp klinik bukak pukul 9am,jadi kami pi breakfast dulu kt Kopitiam berhampiran.Selesai makan terus zasss ke klinik berkenaan.
Kt dlm klinik tu ada byk mainan,jadi sajalah bagi ihsan duduk kt mainan kuda,suka sgt ihsan.Sementara nk tunggu turn kami,ihsan tgk plak ada dua org kanak2 yg agak besar dari dia main ape ek nama mainan tu xingat plak.Bukan main suka ihsan tgk abang dan kakak tu smpai gelak2 terkekek padahal diorang tak kacau pun ihsan.Tiba2 turn ihsan jumpe Dr,xingat plak nama Dr ni tapi Dr pompuan la,baik sgt lembut je.
Lepas dah terangkan mslh ihsan tu Dr check ihsan and the terus nk amik darah nk tgk ape mlsh ihsan.So time nk amik darah ihsan biasalah sure nangis.Tp aku respect ah Dr ni sbb dia amik darah ihsan kejap je n tak bengkak pun.Tp ihsan tetap nangislah kan for sure.Dr suh tunggu kat luar sementara tunggu result.Kami pun tunggu la kt luar,aku kata kt husben td gelak2 ihsan skang kan dh nangis.Pastu aku pun susukan ihsan,naseb baik kt situ ada bilik utk menyusu.
Tak lama lepas tu nurse panggil kami.Dr kata hemoglobin ihsan low 7.4.Dr kata kalu ibu menyusu ramai je yg datang anaknye hemoglobin low dlm 5 cmtue tp sbb ihsan ada bruises so Dr suh bawak pi HUKM terus.Dr terus buat surat.
Aku pun terus call HUKM (tingkat 7) nk tnye ada tak Dr hari ni utk jmpe ihsan.Nurse tu tnye mcm2 ape sakit ihsan sumer,last dia kata nnt jmpe Prof Asma dlm pukul 1245pm sbb dulu ihsan pernah masuk wad under Prof Asma atas mslh cervical lymphoside (betul ke eja?) nye tuh.Sblm pi sana smpat jugak aku singgah ke Butik Ariani yg berdekatan past uterus singgah umah mak jap sbb dekat je dari umah mak nk pi HUKM tu.
Dalam 1215pm kami pun gerak dari umah mak.Terus naik ke tingkat 7 n tunggu utk jmpe Prof Asma.Dlm pukul 1pm baru dpt jmpe nasib Prof Asma sudi meluangkan masa utk tgk ihsan.TQ Prof Asma,.Lepas tgk2 ihsan Prof pun kata “kalu sy kata ihsan kena masuk wad….aku pun sengih je la kt husben sbb masa dlm keta ada tercakap mcm tu,kalu ihsan kena masuk wad cmne….alih2 betul la pulak.Prof Asma nk call Prof B tp mcm takut2 je,katanye PAED ni time lunch hour xangkat call so Prof sms je dulu.Aku mcm pelik Prof Asma cm takut je ngan Prof B rupe2nye semua nurse pun takut gak kt Prof B sbb dia HOD kt PAED bawah tu.huhu.
Then after Prof B balas sms Prof Asma terus Prof Asma call n bgtau condition ihsan.Tp Prof B nk ihsan masuk wad kt bawah bukan kt atas sbb hari sabtu/ahad tak de Dr nk tgk kt tingkat 7 tuh.emmm…so aku ngan husben pun ok je la demi kesihatan ihsan.Jadi hari itu sekali lagi ihsan kena masuk,Bila dh keluar dari bilik Prof jam pun dh masuk waktu zohor terusla husben ke surau bawah utk solat jumaat,tinggallah aku ngan ihsan kt situ sementara tunggu husben ku.
Dlm pukul 2pm husben pun dtg balik,aku kata nurse kata pi daftar dulu kt bawah,tp xde katil kosong lagi.So aku decide nk balik umah dulu kemas2 barang sumer sbb xtau baper lame plak ihsan nk kena tahan kt wad ni.
Dalam pukul 515pm kami pun smpai kt WAD 4A,katil 9.Bermulalah episode baru ihsan di sini.Ptg tu mak n abah pun ada dtg.Husbenku pulak dh gi kelas kt UPM itupun dlm pukul 630pm dia baru gerak.Dlm pukul 7pm mak n abah pun balik.Tinggal la ku berdua ngan ihsan kt sini.Dalam pukul 7 lebih ada sorang Dr pompuan datang. Dr Azlynn ke xsilap namanye mcm2 dia tnye pasal ihsan ni.Pening kepalaku nk menjawab mcm pesalah la pulak nasib ihsan mood ok masa tu.Dr check ihsan pun pening sbb ihsan xlalu diam kaki tangan tiba2 Dr dpt call emergency so dia terpaksa gi dulu.
Tak lama ihsan nenen n tido.Datang pulak Dr lelaki katanye nk tnye mcm2 la.Dr tu tnye mcm2 gak mcm Dr tadi tu tapi kali ni dia tulis balik dlm record ihsan.Pastu tak lama dating pulak 2-3 orang Dr sama gak tnye mcm2 pasal ihsan n check lebam2 ihsan tu.Dr tu suh amik darah ihsan malam ni gak utk tgk ape mlsh nye.Pastu dlm pukul 10pm aku sms husben tnye tak nak dtg ke,aku lapar nk makan.So husben pun dtg bawak makanan.
Aku asyik dengar bunyi baby nangis tp aku xtau dari mana,pastu husben ku kata kt sblh ni baby tux de orang tgk pun.Yeke aku kata???terus aku pi sblh mmg baby tu yg nangis sian sgt aku tgk,dh la asma batuk plak tu.Kakak yg sebelah katil ngan baby tu bgtau ni anak kebajikan xde mak ayah.Ya ALLAH siannye aku ngan baby yg diberi nama ARISYA ini.Terseksa aku tgk dia batuk,aku rasa mungkin dia lapar.Tapi xada nurse yg dtg tgk sbb malam tu nurse ada 3 orang je pastu ada case emergency plak.Aku dukung la arisya kejap,dia diam.Pastu aku bagi dia mnm air masak guna botol ihsan tgk kalu2 dia lapar.Mmg dia lapar,sian sgt.Aku suh husben jaga arisya kejap sbb aku nk pump susu bagi kat arisya.Alhamdulillah ada rezeki arisya utk mnm susu aku.Syukur padamu ALLAH yg maha kaya.Lps tu dia diam maybe sbb da kenyang.Aku rasa dh lewat malam baru nurse amik arisya tukar pampers n bagi susu tp arisya mcm dh tak nak sbb tadi dia dh dpt susu ibu.
Pastu dlm pukul 11 lebih ke husben balik dia takut nak tdo sini esok malam baru tdo ye lah takut kena marah ngan nurse.Then dlm pukul 12 lebih Dr panggil nk amik darah ihsan.Aku tau sure ihsan menjerit2 la lepas ni.Sian sgt kt ihsan.Okla nnt aku smbg lagi sbb panjang lagi kisah di WAD 4A sepanjang 7 hari kami berkampung kt sini.
Friday, April 16, 2010
Hemophilia A (Factor VIII Deficiency)
Hemophilia A
Hemophilia A is the most common type of hemophilia. It is also known as factor VIII deficiency or classic hemophilia. It is largely an inherited disorder in which one of the proteins needed to form blood clots is missing or reduced. In about 30% of cases, there is no family history of the disorder and the condition is the result of a spontaneous gene mutation.
Approximately one in 5,000 males born in the United States has hemophilia. All races and economic groups are affected equally. When a person with hemophilia is injured, he does not bleed harder or faster than a person without hemophilia, he bleeds longer. Small cuts or surface bruises are usually not a problem, but more traumatic injuries may result in serious problems and potential disability (called "bleeding episodes"). There are different levels of hemophilia: mild, moderate, and severe: • People with mild hemophilia (6% to 49% factor level) usually have problems with bleeding only after serious injury, trauma, or surgery. In many cases, mild hemophilia is not discovered until an injury or surgery or tooth extraction results in unusual bleeding. The first episode may not occur until adulthood. • People with moderate hemophilia, about 15% of the hemophilia population, tend to have bleeding episodes after injuries. They may also experience occasional bleeding episodes without obvious cause. These are called "spontaneous bleeding episodes." • People with severe hemophilia, about 60% of the hemophilia population, have bleeding following an injury and may have frequent spontaneous bleeding episodes, often into the joints and muscles.
Everyone inherits two sex chromosomes, X and Y, from his or her parents. A female inherits one X chromosome from her mother and one X chromosome from her father (XX). A male inherits one X chromosome from his mother and one Y chromosome from his father (XY). The gene that causes hemophilia is located on the X chromosome.A woman who gives birth to a child with hemophilia often has other male relatives who also have hemophilia. Sometimes, a baby will be born with hemophilia when there is no known family history. This means either that the gene has been "hidden" (that is, passed down through several generations of female carriers without affecting any male members of the family) or the change in the X chromosome is new (a "spontaneous mutation"). There are four possible outcomes for the baby of a woman who is a carrier. These four possibilities are repeated for each and every pregnancy: 1. A girl who is not a carrier 2. A girl who is a carrier 3. A boy without hemophilia 4. A boy with hemophilia With each pregnancy, a woman who is a carrier has a 25% chance of having a son with hemophilia. Since the father's X chromosome determines the baby will be a girl, all the daughters of a man with hemophilia will be carriers. None of his sons, which is determined by the father through his Y chromosome, will have hemophilia.
Genetic counseling is available at most HTCs. These professionals have information to help you make family planning decisions. In general, small cuts and scrapes are treated with regular first-aid: clean the cut, then apply pressure and a band-aid. Individuals with mild hemophilia can use a non-blood product called desmopressin acetate (DDAVP) to treat small bleeds. Deep cuts or internal bleeding, such as bleeding into the joints or muscles, require more complex treatment. The clotting factor missing (VIII or IX) must be replaced so the child can form a clot to stop the bleeding. Some factor products are made from human blood products such as donated plasma. Others, called "recombinant factor," are made in a laboratory and do not use human blood products. The Medical and Scientific Advisory Council of the National Hemophilia Foundation encourages the use of recombinant clotting factor products because they are safer. Your doctor or your HTC will help you decide which is right for you. All factor treatments are injected or infused directly into the veins. In cases of severe hemophilia, doctors sometimes recommend giving a regimen of regular factor replacement treatments (a therapy called prophylaxis) to prevent bleeding episodes before they happen. The Medical and Scientific Advisory Council of the National Hemophilia Foundation recommends prophylaxis as optimal therapy for children with severe hemophilia A and B.
Notify your doctor or HTC if your child does not respond to the usual dose of factor. In rare instances, people can develop an inhibitor, to standard factor treatment. In the event this occurs, your doctor or HTC will work with you to develop a special plan of care.
Hemophilia A is the most common type of hemophilia. It is also known as factor VIII deficiency or classic hemophilia. It is largely an inherited disorder in which one of the proteins needed to form blood clots is missing or reduced. In about 30% of cases, there is no family history of the disorder and the condition is the result of a spontaneous gene mutation.
Approximately one in 5,000 males born in the United States has hemophilia. All races and economic groups are affected equally. When a person with hemophilia is injured, he does not bleed harder or faster than a person without hemophilia, he bleeds longer. Small cuts or surface bruises are usually not a problem, but more traumatic injuries may result in serious problems and potential disability (called "bleeding episodes"). There are different levels of hemophilia: mild, moderate, and severe: • People with mild hemophilia (6% to 49% factor level) usually have problems with bleeding only after serious injury, trauma, or surgery. In many cases, mild hemophilia is not discovered until an injury or surgery or tooth extraction results in unusual bleeding. The first episode may not occur until adulthood. • People with moderate hemophilia, about 15% of the hemophilia population, tend to have bleeding episodes after injuries. They may also experience occasional bleeding episodes without obvious cause. These are called "spontaneous bleeding episodes." • People with severe hemophilia, about 60% of the hemophilia population, have bleeding following an injury and may have frequent spontaneous bleeding episodes, often into the joints and muscles.
Everyone inherits two sex chromosomes, X and Y, from his or her parents. A female inherits one X chromosome from her mother and one X chromosome from her father (XX). A male inherits one X chromosome from his mother and one Y chromosome from his father (XY). The gene that causes hemophilia is located on the X chromosome.A woman who gives birth to a child with hemophilia often has other male relatives who also have hemophilia. Sometimes, a baby will be born with hemophilia when there is no known family history. This means either that the gene has been "hidden" (that is, passed down through several generations of female carriers without affecting any male members of the family) or the change in the X chromosome is new (a "spontaneous mutation"). There are four possible outcomes for the baby of a woman who is a carrier. These four possibilities are repeated for each and every pregnancy: 1. A girl who is not a carrier 2. A girl who is a carrier 3. A boy without hemophilia 4. A boy with hemophilia With each pregnancy, a woman who is a carrier has a 25% chance of having a son with hemophilia. Since the father's X chromosome determines the baby will be a girl, all the daughters of a man with hemophilia will be carriers. None of his sons, which is determined by the father through his Y chromosome, will have hemophilia.
Genetic counseling is available at most HTCs. These professionals have information to help you make family planning decisions. In general, small cuts and scrapes are treated with regular first-aid: clean the cut, then apply pressure and a band-aid. Individuals with mild hemophilia can use a non-blood product called desmopressin acetate (DDAVP) to treat small bleeds. Deep cuts or internal bleeding, such as bleeding into the joints or muscles, require more complex treatment. The clotting factor missing (VIII or IX) must be replaced so the child can form a clot to stop the bleeding. Some factor products are made from human blood products such as donated plasma. Others, called "recombinant factor," are made in a laboratory and do not use human blood products. The Medical and Scientific Advisory Council of the National Hemophilia Foundation encourages the use of recombinant clotting factor products because they are safer. Your doctor or your HTC will help you decide which is right for you. All factor treatments are injected or infused directly into the veins. In cases of severe hemophilia, doctors sometimes recommend giving a regimen of regular factor replacement treatments (a therapy called prophylaxis) to prevent bleeding episodes before they happen. The Medical and Scientific Advisory Council of the National Hemophilia Foundation recommends prophylaxis as optimal therapy for children with severe hemophilia A and B.
Notify your doctor or HTC if your child does not respond to the usual dose of factor. In rare instances, people can develop an inhibitor, to standard factor treatment. In the event this occurs, your doctor or HTC will work with you to develop a special plan of care.
Monday, April 5, 2010
Pusat Pemuliharaan Gajah,Kuala Gandah
Gajah2 di Pusat Pemuliharaan Gajah,Kuala GandahPusat Konservasi Gajah Kebangsaan Kuala Gandah
Pusat Konservasi Gajah Kebangsaan Kuala Gandah telah ditubuhkan oleh Jabatan Perlindungan Hidupan Liar dan Taman Negara (PERHILITAN) pada tahun 1989. Pusat ini merupakan ibu pejabat Pasukan Pemindahan Semula Gajah yang telah wujud sejak tahun 1974. Pusat ini juga merupakan tempat perlindungan bagi beberapa ekor gajah denak dan liar. Pusat Pemuliharaan Gajah Kebangsaan Kuala Gandah juga mempunyai matlamat untuk meningkatkan kesedaran di kalangan orang ramai tentang ancaman dan tekanan yang dihadapi oleh gajah di Malaysia. Selain itu, pusat pemuliharaan ini menggalakkan kajian dan penyelidikan berkaitan teknik pemindahan semula dan konservasi gajah. Pusat ini mempunyai beberapa ekor gajah tempatan yang dibawa masuk daripada Thailand dan Myanmar. Gajah-gajah ini dilatih dan digunakan untuk memindahkan gajah-gajah liar dari kawasan-kawasan yang bermasalah di seluruh Semenanjung Malaysia. Pusat ini juga memelihara anak-anak gajah yang ditinggalkan oleh ibunya untuk memastikan anak-anak gajah yang ditinggalkan oleh ibunya terus hidup.
Info Pelawat : Jadual Aktiviti Pelawat
Masa Aktiviti
2.15 petang MENUNGGANG GAJAH
2.30 petang MEMANDIKAN GAJAH
3.30 petang MEMBERI MAKANAN
3.45 petang VIDEO DOKUMENTARI
Untuk maklumat lanjut, sila hubungi : Ketua Pusat Konservasi Gajah Kebangsaan Kuala Gandah Jabatan PERHILITAN Kuala Gandah, 28500 Lanchang Pahang Darul Makmur, Malaysia
Tel: 09-279 0391
Pautan berkaitan : Laman Web Rasmi PERHILITAN • http://www.wildlife.gov.my/bhg_kualagandah.html
Untuk maklumat lanjut, sila hubungi : Ketua Pusat Konservasi Gajah Kebangsaan Kuala Gandah Jabatan PERHILITAN Kuala Gandah, 28500 Lanchang Pahang Darul Makmur, Malaysia
Tel: 09-279 0391
Pautan berkaitan : Laman Web Rasmi PERHILITAN • http://www.wildlife.gov.my/bhg_kualagandah.html
Thursday, April 1, 2010
Program Amal Kuala Gandah
Salam...hari ni just inform that insyaALLAH sabtu ni kami sekeluarga akan joint program amal kuala gandah anjuran SARAKIDS....so kepada kawan2 semua yg mungkin nk menderma bekalan makanan/baju2 lama/or whatever utk diberi kepada Org Asli kt sana boleh bagi terus kepada SARAKIDS or boleh juga kirim pd kami sekeluarga.Bekalan makanan utk anak2 gajah juga dialu2kan.Ape2 sy akan update aktiviti di sana nnt....see ya...
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